Vascular access steal syndrome is a syndrome caused by ischemia (not enough blood flow) resulting from a vascular access device (such as an arteriovenous fistula or synthetic vascular graftâÂÂAV fistula) that was installed to provide access for the inflow and outflow of blood during hemodialysis.
Symptoms are graded by their severity:
The fistula flow can be restricted through banding, or modulated through surgical revision.
If the above methods fail, the fistula is ligated, and a new fistula is created in a more proximal location in the same limb, or in the contralateral limb.
While banding techniques such as MILLER banding apply external constriction to the outflow vein post-implantation to reduce excessive flow in arteriovenous (AV) accesses, some patented designs incorporate flow-restrictive elements directly into the AV graft or stent during construction or deployment. These create a controlled stenosis or reduced-diameter section to regulate blood flow, create a pressure drop, preserve distal perfusion, and mitigate complications such as steal syndrome, venous hypertension, and excessive cardiac demandâÂÂsimilar in hemodynamic effect to external banding but integrated into the device itself.
Examples include patents by Stanley Batiste describing AV dialysis grafts with built-in flow restrictions:
Such integrated designs may simplify management of high-flow accesses by embedding restriction features, though clinical outcomes, adoption, and direct comparisons to post-implant banding procedures like MILLER remain device-specific and variable.
DASS occurs in about 1% of AV fistulas and 2.7-8% of PTFE grafts.
Within the contexts of nephrology and dialysis, vascular access steal syndrome is also less precisely just called steal syndrome (for short), but in wider contexts that term is ambiguous because it can refer to other steal syndromes, such as subclavian steal syndrome or coronary steal syndrome.